Lens‑particle glaucoma and inflammatory glaucoma are secondary glaucomas in which trabecular outflow is impaired by lens material or inflammatory debris. Both can cause raised pressure and optic nerve damage if not recognised promptly.
Lens-particle Glaucoma
Lens‑particle glaucoma occurs when lens fragments enter the anterior chamber after cataract surgery or trauma; these particles obstruct the trabecular meshwork and provoke inflammation.
- Symptoms include pain, redness, and blurred vision after lens disruption.
- Treatment uses pressure‑lowering drops, anti‑inflammatory therapy, and removal of residual lens material.
- Timely washout usually restores pressure control.
- Persistent cases may need glaucoma surgery.
Inflammatory Glaucoma
Inflammatory (uveitic) glaucoma arises when uveitis‑related cells, protein, or scarring interfere with aqueous drainage or when steroids used to treat inflammation raise pressure.
- Management balances controlling inflammation with minimising steroid‑induced pressure rise.
- Pressure‑lowering drops, systemic immunosuppression, and sometimes surgery are required.
- Angle synechiae and trabecular damage can make long‑term control challenging.
- Regular monitoring during and after uveitis flares is essential.
