From a strictly clinical viewpoint, most CMO responds well to treatment and eventually allows good visual acuity.¹
For patients, however, the experience of delayed recovery can be deeply frustrating, especially when they chose private surgery for speed and predictability. Blurred central vision, fluctuating clarity and the need for prolonged drops can undermine confidence in the whole process.
That is why we treat CMO prevention as both an anatomical and experiential priority. By combining universal OCT, extended steroid–NSAID prophylaxis and early-warning surveillance, we aim not only to keep the macula dry but also to maintain the smooth recovery patients expect.
When CMO does occur, fast recognition and clear explanation help restore trust and minimise anxiety.² Transparency about risks, mechanisms and our own data underpins every stage of this conversation.
References
- Lobo C. Pseudophakic cystoid macular edema. Ophthalmologica. 2012;227(2):61-67.
- Henderson BA, Kim JY, Ament CS, et al. Clinical pseudophakic CMO risk factors. J Cataract Refract Surg. 2007;33(9):1550-1558.
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